Sunday, March 11, 2018

Dollars for Docs

Pharmaceutical and medical device companies are now required by law to release details of their payments to a variety of doctors and U.S. teaching hospitals for promotional talks, research and consulting, among other categories. Use this tool [link below] to search for general payments (excluding research and ownership interests) made from August 2013 to December 2015.

The following link includes payments from 2013 to 2015

Dollars for Docs

The following link includes payments made through 2016:

Open Payments Data

Other Links:

About the Dollars for Docs Data
ProPublica’s Dollars for Docs database contains payments to doctors and teaching hospitals from pharmaceutical and medical device companies made between August 2013 and December 2015. The disclosures were required under the Physician Payments Sunshine Act, a part of the 2010 Affordable Care Act.

The database includes “general payments” — 15 categories including promotional speaking, consulting, meals, travel and royalties. It does not include research payments nor does it include physicians’ ownership stakes in companies. Research payments will be included in Dollars for Docs in the future. Detailed descriptions of the payments can be found here.

The doctors included in our tool include medical doctors (MD), dentists, osteopaths (DO), optometrists, podiatrists and chiropractors. The tool does not include nurse practitioners and physician assistants (because companies are not required to report payments to them.) The tool also allows you to search teaching hospitals.

We’ve taken the payment reports, which were released by the Centers for Medicare and Medicaid Services, and compiled them into a single, comprehensive database that allows patients to search for their physician and receive a listing of all payments matching that name. We provide rankings for each doctor to allow comparisons to peers in the same specialty and state.

Our analysis found that most doctors take payments, and that doctors who receive payments are, on average, more likely to prescribe a higher percentage of brand-name drugs. For each physician in Dollars for Docs, we document the number of payments he or she received, the total of those payments, and the number of different companies that paid him or her.

The bulk of each physician’s page is taken up by information on each payment he or she received, the company making the payment, the date of the payment, the names of the drugs and medical devices associated with the payment, and whether the payment was made to a third party entity. Sometimes, payments are not made directly to doctors but instead are provided to their universities, medical practices or research centers. We also note whether a doctor has disputed the payment.

By default, a physician’s page shows aggregate amounts received by year and details of payments in the most recent year reported. Users can display other years by using the dropdown at the top of each doctor page. Company and product pages display aggregate totals across all payment years (2013 to 2015).

Sometimes, more than one company makes payments related to a single drug or device. On each product page, we note the number of companies making payments, as well as the names of those companies. Each payment can also be attributed to more than one product, so we note the percentage of payments that relate only to that product (meaning no other products were mentioned in the payment).
Use the Data

Get the data that powers this investigation. A digital download is available for purchase in the Data Store.

ProPublica has published Dollars for Docs since 2010, at first using payment reports that certain companies were required to publish as part of legal settlements with the federal government. Often, these settlements were related to whistleblower lawsuits alleging improper marketing or kickbacks by the company. The now-archived version of our database includes $4 billion in payments from 17 companies, from 2009 to 2013. An archive of those payments is still available at

We have made some effort to normalize the data and eliminate duplicates, but data is primarily as it has been reported by the companies to the Centers for Medicare and Medicaid Services.

About Open Payments
Open Payments is a national transparency program that collects and publishes information about financial relationships between the health care industry (i.e. drug and device companies) and providers (i.e. physicians and teaching hospitals). These relationships may involve payments to providers for things such as research, meals, travel, gifts, or speaking fees. One of the ways that the Centers for Medicare & Medicaid Services (CMS) provides data to the public is through this search tool, which allows the public to search for physicians and teaching hospitals receiving payments, as well as companies that have made payments.

The purpose of the program is to provide the public with a more transparent healthcare system. All information available on the Open Payments database is open to personal interpretation and if there are questions about the data, healthcare consumers should speak directly to the healthcare provider for a better understanding. More information about the program can be found on the CMS Open Payments website.

There you can get an overview of the data that is collected and displayed and learn more about what is included in the data. For other Open Payments related questions, contact the Open Payments team at


Thursday, March 01, 2018

Liberty and Justice for All?

With more than two million people behind bars, the United States has the world's largest prison population, a 500% increase over the last 40 years.  Changes in law and policy, not changes in crime rates, explain most of this increase. In fact, crime rates have decreased. The U.S. also has the second-highest rate of incarceration and that doesn't include the more than six million, or almost 3% of the voting population, who are disenfranchised due to past convictions, felony disenfranchisement.    Nor does it include over 23 million widely stigmatized people, the "vast underground army of released felons — adult men and women convicted of serious criminal offenses for which they have been punished with prison time or probation, and who now form part of the general population.

Click image to enlarge


Prison Policy Initiative

The Sentencing Project

Lawsuit reveals how tech companies profit off the prison-industrial complex

The end of American prison visits: jails end face-to-face contact – and families suffer


Sunday, January 21, 2018

Are We Raising Our Children to Be Slaves?

You can’t make it without an education!" "Go to college!" "Graduate from high school, go to college, get a good job." How many of us heard these mantras while growing up? How many of us have said these mantras to our children? Other people's children? Despite the fact that getting a  college degree today paves the road to debt slavery  where the chains and fetters are invisible and the wealthy, powerful owners, anonymous.

Slavery is likely to be abolished by the war power and chattel slavery destroyed. This, I and my European friends are glad of, for slavery is but the owning of labor and carries with it the care of the laborers, while the European plan, led by England, is that capital shall control labor by controlling wages." -- Chas. Hazzard, The Hazzard Circular, 1862
As we start 2018, student loan debt is close to $1.5 trillion, where an estimated 44.2 million Americans have one or more student loans on file.  Student loan debt surpasses total U.S. credit card debt by approximately $659 billion. It is the second largest financial asset on the federal government's balance sheet, making up 51.8% of total assets. Only mortgage debt is higher. Not to mention, according to the Federal Reserve is the only form of consumer debt that continued to grow in the wake of the Great Recession.

Parents and students have little understanding of how this loan program operates.   They don't understand that there is an entire "ecosystem feeding on federal student loans."
The companies making those calls are just one part of an ecosystem feeding on federal student loans. There are also debt servicers, refinance lenders, firms that help former students stay out of default and for-profit schools that make money as borrowers try to repay more than $1.2 trillion in government-backed education debt.
They don't understand that the cost of living is rising faster than income that make it increasingly difficult for people to keep up with the everyday expenses of life. They don't understand that a four-year degree is no longer the golden ticket to full time job with benefits and job security. They don't understand that the "four-year" degree actually takes five, six or even more years, which, of course, costs even more money.

When will we catch up with reality?  What was true 40 years ago doesn't make it true today.  What was true for baby boomers is not true for millennials and/or generations x,y,z, etc. 
It’s not unusual for me to talk to a couple that between them has $200,000 in student loan debt now. It’s not unusual at all. I talk to them almost every day on this show. And they’re 32 years old, they’ve been out of school for four, five or six years and they’re just treading water. They’re stuck because no one in their life—no supposed high school counselor, no parent, no financial-aid officer—smack you silly. Financial aid is $200,000 in student loan debt. Give me a break. Nobody looked at you and said, “You know, when you bring that baby home from the hospital, there’s a possibility you may want to use your education to raise your children.” -- Dave Ramsey


Student Debt Crisis

Hazard Chronicle Documentary Evidence

Student Debt Relief

Student Debt Slavery: Bankrolling Financiers on the Backs of the Young

Narrow bankruptcy laws make it nearly impossible to discharge student debt: From the The Cost of Opportunity: A series chronicling the student loan debt crisis in Wisconsin series

Rising Tuition Costs and the History of Student Loans


Tuesday, October 24, 2017

War on Health: Profits Before Patient Safety

Keep in mind, according to CDC statistics, no one has died from the use of food or dietary supplements, yet now, thanks to the FDA, you’ve got 3x as much regulation for food and dietary supplements as you have for pharmaceutical drugs, despite the fact that pharmaceutical drugs have proved to be one of the top killers of American citizens.

The relationship between physicians and the drug industry doesn’t begin once you have your MD or once you own your private practice; it begins the day you hit medical school. Big Pharma often give medical students gifts on their very first day. These gifts are always “Big Pharma” propaganda disguised as education, or something medical industry related that leaves the hard pressed medical student feeling like, at least someone cares, because we all know how tough medical school is.

But why is it so tough? Why are there intern boot camps?  Why must medical students and residents go through Why is "the private group that oversees physician training in the United States proposed rolling back rules so that young doctors just out of medical school can work shifts as long as 28 hours"? I mean, who wants an exhausted medical student/intern/resident practicing their craft on you when you are at your most vulnerable? Well one answer is that "medical school functions as a highly efficient system of inDOCtrination to ensure that physicians are less likely to question or confront the systems of power."
Professors who have already been indoctrinated to think a particular way ensure discussions are kept "on topic," or within the traditional bounds of acceptable debate that do not challenge power. I personally have lost track of the number of times I have heard a professor say, "That is interesting, but it is just outside of the scope of the discussion we are trying to have." This is a highly efficient and subtle way of controlling thought.

It makes more sense when you consider modern medicine was founded by a robber barons and an oil tycoon in particular, John D. Rockefeller. The conspiracy to limit and eliminate competition from non-drug therapies began with the Abraham Flexner Rockefeller Report on Medical Education of 1910. Abraham Flexner was engaged by John D. Rockefeller to  “evaluate” the effectiveness of therapies taught in medical schools and other institutions of the healing arts. The Flexner report unequivocally recommended the closure of all the homeopathic and naturopathic medical schools, in other words, anyone or any institution who use natural medicines to heal.

Subsequently, federal alphabet agencies--FDA, CDC, etc.-- were created to enforce the allopathic model under the guise of "protecting" the American food and drug supply.   In actuality, these agencies serve the medical industrial complex, not American citizens as you will see in the following documentaries.  The reason is obvious: alternative, mostly inexpensive non-toxic therapies represent a potential loss of billions, if not trillions of dollars to allopathic (drug) medicine and drug companies, not to mention, there is nothing more threatening to elite power than a healthy, robust public.


Overdosed America: The Broken Promise of American Medicine by John Abramson, M.D.

AIDS, Opium, Diamonds, and Empire: The Deadly Virus of International Greed. by Nancy Turner Banks, M.D.

Treaties and International Agreements


Wednesday, October 04, 2017

Las Vegas Shooting: Cui Bono?

I don't claim to know the truth about the Las Vegas incident, but I do know that like most lone gunman mass shooting events that turn into giant 24/7 media circuses, the official story just doesn't make sense even before you scratch the surface.

I mean, the guy carried 23 guns to his room and none of the security cameras caught it? Where is the camera footage of him prior to the shooting, carrying all of his weapons? Casinos literally hire former Marines with sharpshooter training in addition to the incredible surveillance of every public area, not to mention 22,000 people with smart phones. We should be awash with CCTV and cell phone footage of the chaos an event like this would create. 22,000 people with smart phones and so little , didn't smart-phone video this. Dig even deeper and the story gets even more ludicrous, preposterous, and should be downright offensive to anyone who spends five minutes on the official narrative.

High Incident Project?

Cui Bono? It certainly wasn't the alleged patsy shooter. Who benefits? An insidious industry that reaps maximum profit and influence for all the institutions that live off fear: the police, the justice system, the media, the weapons and security trade and most importantly, government, who create the "Big Brother" laws that increases their power over the masses. In other words, government and private contractors who have a stake in an exponentially growing national security state from which they stand to make a fortune and further limit our freedom of movement.

Collateral damage to push hidden agendas? Seems that way.


Sunday, September 24, 2017

Organ Harvesting: Dissected Alive for Profit?

Most of us sign up to become organ donors, literally, out of the goodness of our hearts, but I've always been a little skeptical (fear of my death being hastened by an eagerness to procure my organs) even before I knew the truth: that our vital organs, in order to be viable for transplant, must be harvested from human beings with a pulse, in other words, alive.

You say, how can that be? What part of after-I'm-dead don't they understand?  Well, you must ask yourself: "What is the legal definition of death?" "What was the legal definition of death?" "Why did the legal definition of death change?" "If a determination of death must be made in accordance with "accepted medical standards", how are "accepted medical standards" created and what are they based on?" "What happened to the great principle, both medical and moral, that governs the medical profession: Primum, non nocere (First, do no harm)?"

In a nutshell, in order for organ transplantation to occur, the need for "dead patients with live organs" became paramount.Below, is an attempt to answer some of those questions, while exposing the truth about organ donation; however, I implore you to do your own research.

The common law standard of death used to be total cessation of cardiac and respiratory function; that is, observable signs that life has ended.  Unfortunately, that  clear definition of death renders organ transplantation, impossible. Yet, human organ transplantation is a multi-billion dollar industry. What gives?  What gives is the legal definition of death. The medical industrial complex, specifically the 1968 Ad Hoc Harvard Committee on Irreversible Coma, created in response to the development of organ transplantation redefined the end of life in such a way that made the transplantation of organs possible.  So, the legal definition of death became subjective, a matter of opinion.

From the book, Death Investigation in America: Coroners, Medical Examiners, and the Pursuit of Medical Certainty By Jeffrey M Jentzen

The proponents of organ donation--transplant surgeons and bioethicists--represented two powerful new specialties. A decades long conflict began between these specialists and medical examiners, coroners, and prosecutors over the major basis of the latter's professional status--the possession of the body. Now, a lucrative business of organ procurement and a sentimental public relations campaign pressing for organ transplantation began to criticize medical examiners. Created in part in response to the development of organ transplantation, the 1968 Ad Hoc Harvard Committee on Irreversible Coma established that an individual could be legally pronounced dead by the physician based on the constellation of clinical findings that did not include the heart ceasing to beat.  Against aggressive organ procurement agencies, for profit, tissue recovery teams, and transplant surgeons entered the fight for desperate patients in a battle over possession of the body. Coe and his colleagues faced new challenges in having to wrestle with the sometimes conflicting goals of their obligation to investigate death and their responsibility to supply the public's increasing demand for organs and tissues to support life.

The definition of what constituted death changed from the absence of observable sign of breathing, movement and heartbeat to more subtle definitions based on lack of blood flow to the brain."
Now that the legal determination of death, since the advent of "cadaver" organ transplantation, has eliminated the total cessation of cardiac and respiratory function, death is now defined as the cessation of brain function--brain death--but, what is brain death? How can you be completely dead if your heart is still pumping, if your lungs are still working? Keep in mind, ventilators do not breathe for the patient, they only force air into the lungs; the patient must breathe out on his or her own.  And the diagnosis of Persistent Vegetative State (PVS) is incorrect almost half the time.  In other words, there is no true standard, and the diagnosis of brain death is completely subjective, a matter of opinion based on hospital  policy that is often influenced by profit margin.

But how do they determine "brain death"? Well, there is no legal or even statewide standard State law varies greatly and hospitals dictate what tests (if any) are used for the diagnosis. Many states allow nurses or nurse practitioners to declare brain death. However, there is one test that stands out above the rest and that is the Apnea Test.

Doctors and hospitals proclaim that an Apnea test can be used to confirm brain death. It does no such thing. In fact, it only makes things worse for the patient, your loved one. During the Apnea test, they take the ventilator away for up to ten minutes, at a time when the patient needs oxygen the most, to see how the patient responds. Without that crucial oxygen, the carbon dioxide level goes up which makes the brain swell, making it much more likely the patient will experience true brain death. Once again, keep in mind that a ventilator will not work on a dead person. The only thing a ventilator does is push air in. It does not push air out. Despite what they tell you, the ventilator only works when the lungs, heart, kidneys, liver are functioning. In other words, the patient is not dead!

Now, if you choose to opt out, it's not simply a matter of unchecking the little box on your driver's license or informing family members that should something happen to you, you do not want to donate your organs. Thanks to the Uniform Anatomical Gift Act, adopted in 48 states, you must have a document of refusal to opt out.
donor cards are legally binding in 48 states and health professionals who act on them are immune from liability in every state."
The revised UAGA (2006) reaffirms that if a donor has a document of gift, there is no reason to seek consent from the donor family as they have no right to give it legally.  If an individual has not made a document of gift during life, the Revised UAGA (2006) presumes the intent to donate organs, therefore has expanded the list of person (in section 9a  who can consent on behalf of the individual...Finally, if an individual prefers not to donate, this must be documented in a signed, explicit refusal."

It's a good idea to do your homework and research the evolution of major laws that make organ transplantation possible. The major ones are Uniform Determination of Death Act (UDDA) created in 1981, and the Uniform Anatomical Gift Act (UAGA), not to mention the Dead Donor Rule, which isn't exactly a law, but a general ethical assumption, enshrined in the UDDA.
The 1998 DHHS Referral and Request Regulation.

In light of the current problems regarding the lack of supply of suitable cadaveric organs, on December 15, 1997, Vice President Al Gore along with the DHHS launched a national initiatives to increase organ donation by 20%.

One element of the national initiative was to propose a rule ensuring that next-of-kin are asked to consent to the procurement of their loved ones organs. As a result, the DHHS passed a Referral and Request Regulation in August of 1998. 

The new regulation provides that hospitals wishing to receive Medicare payments must refer their patients who died along with their patient whose deaths are imminent to a local Organ Procurement Organization (OPO). Consequently, the OPO would provide personnel trained and experienced in obtaining consent to consult with the patient's next of kin and request consent to procure their loved ones organs.

At the same time that an increasing amount of research is finding the brain can heal itself, the use of aggressive tactics by organ procurement teams towards families to accept a diagnosis of "brain death" is increasing.  The result is that the diagnosis of "brain death" has been rapidly increasing over the past several years.  There is no doubt that lucrative financial outcomes factor in when determining brain death. 

To put it simply, the organ donor card gives doctors your permission (under contract law) to remove your organs from your warm, breathing and UN-anesthetized, doctor-declared “brain-dead” body. Keep in mind that young people with healthy organs  are the best candidates for organ harvesting.

As, Dr. Robert Truog, Professor of Medical Ethics, Anesthesiology and Pediatrics at Harvard Medical School asked,
Is our understanding of the facts driving our conclusion or is our desire for certain conclusions driving our interpretation of the facts? Are we gerrymandering the lines between life and death solely to meet social goals? In the long run, is this more likely to bolster or erode the confidence and trust of the people in the organ donation enterprise? "

P.S. I'm only advocating for fully informed organ donors--informed consent--not the end of organ transplantation. I'm only trying to encourage due diligence and the education of oneself and others on the organ donation program. The bottom line is: Challenge the "brain death" diagnosis. Don't sign off on the hospital's advanced directives.   And above all, do not consent to an Apnea test for loved ones!

Think about it, why do they administer paralyzing drugs and sometimes even anti anxiety drugs--however, no anesthesia-- to the organ donor while operating on them?  Because they're not brain dead! Or any kind of dead!    I woke up in the middle of an operation...all I can tell you is that I've never experienced so much pain.  Thank God I could move and scream to alert the doctors.  My worst nightmare is being operated on, feeling every little move of the surgeon, without the ability to move, scream, alert anyone that I'm being tortured. I imagine I'm not the only one.


Controversies in the Determination of Death: A White Paper by the President's Council on Bioethics

How to revoke organ donation consent by state

Horror as patient wakes up in NY hospital with doctors trying to harvest her organs for transplant profits

An 8-year-old was taken off life support, his organs donated. Now, police are investigating

Full Court Document filed regarding lawsuit regarding coroner's office and 8-year old taken off life support to get his organs

Surgeon Accused of Speeding a Death to Get Organs

Ruben Navarro Civil Case Filing:

Doctor Cleared of Harming Man to Obtain Organs

Can Brain Dead Patients Respond?

"Brain Dead" Patient Begins Breathing During Surgery After Liver Removed and Other Horrific Accounts:
There's a big difference between mostly dead and all dead. Now, mostly dead … is slightly alive."


Friday, September 15, 2017

Immersed In an Ocean of Electromagnetic Radiation

We can't turn on the TV/radio, open a newspaper/magazine/book without being bombarded by warnings about anthropomorphic global warming/climate change (by the way, a multi-billion dollar industry based on psuedoscience), yet we hear nothing about the very real danger of anthropomorphic electrosmog. The earth is completely smothered in man-made electromagnetic radiation.

The pulse of the earth 7.83 Hz. The human alpha waves of the human brain is 7.83 Hz. That's not a coincidence, but the power grid, crisscrossing the country, radio, TV, satellite, pagers, cell phone,wireless networking, etc., surrounding us have almost totally permeated our environment with an invisible electromagnetic attack practically impossible to escape.
In the mid eighties fewer than 3% of all people in most of the countries were using cell phones, but if you fast forward to today almost 100% of the people are using cell phones so the question we should all be asking, "is there anyone protecting the public from the invisible ocean of microwave radiation generated from the trillion dollar telecommunications industry?" The answer is a resounding NO!

Brushing aside smart meters, Wifi, and other forms of electromagnetic radiation, the most popular gadget of our age--the cell phone-- has now been shown to damage DNA, break down the brain's defenses, reduce sperm count, increase memory loss, and dramatically increase the risk of Alzheimer's disease and cancer. The growing brains of children make them especially vulnerable, yet half of the world's four billion cell phone users are under twenty.

So, with the constant smog of man-made frequencies surrounding our daily lives and no testing as to its safety, is it any wonder people's bodies have started to react?"

Measuring Schumann resonance in or around a city has become impossible. Electromagnetic pollution from cell phones has forced us to make our measurements at sea. – Dr. Wolfgang Ludwig, physicist


Disconnect: The Truth About Cell Phone Radiation, What the Industry Has Done to Hide It, and How to Protect Your Family by Devra Davis
Davis, the founding director of the toxicology and environmental studies board at the U.S. National Academy of Sciences, takes listeners through the dark side of this trillion-dollar industry. Health experts have long been frozen out of policy-making decisions about cell phones; federal regulatory standards are set by the cell phone industry itself. Cell phone manufacturers have borrowed the playbook of the tobacco industry. One secret memo reveals their war plan against reports of cell phone dangers. Among a host of fascinating characters, Davis introduces Om P. Gandhi, a world expert on how cell phone radiation penetrates the human brain. Once a consultant to major cell phone companies, Gandhi now refuses to work with them. We also meet Franz Adlkofer, who led the multi-lab study that showed once and for all that brain cell DNA is unraveled by cell phone microwave radiation-and, as Davis dramatically portrays, it nearly cost him his career. As this eye-opening call to action shows, we can make safer cell phones now. Why would we put our children at risk of a devastating epidemic of brain illness in the years to come?


Monday, August 21, 2017

Meet the Should Be Exonerated: Crosley Green

Crosley Green
Update: (August 19, 2017):

Despite a mountain of evidence that proves Crosley Green's innocence, in January 2016, due to a trivial procedural technicality (one day late), Green's petition for a new trial was denied. So, in other words, 59-year old Crosley Green could stay in prison for the rest of his life even though there is very clear evidence of his innocence and very clear evidence that he should never have been convicted in the first place.
"People are alarmed to find out that courts have no problem at all saying you filed one day late… we're gonna use that as a basis to keep you in prison for the rest of your life not withstanding the fact that you can prove a clear miscarriage of justice," -- Seth Miller, runs the Innocence Project of Florida.

And then, in June 2017,the 11th Circuit Federal Court of Appeals will allow Green's attorneys to argue in person why his case should not have been dismissed. If the three-judge panel agrees, Crosley Green will finally get his case heard in federal court.

Please watch the the most recent 48 Hours episode which also features three similar cases of wrongful convictions from Brevard County Florida in the 1980s that were subsequently overturned.

Please sign this petition for clemency (or new trial) on


Something new and different, a Florida man convicted for murder and sentenced to death simply because he was black. As attorney, Keith Harrison said, "an example of race being a substitute for evidence,race substituted for evidence.

For more than 16 years, 48 Hours has investigated the case of the 1989 murder of 22-year old Charles "Chip" Flynn because they believed it involved prosecutorial misconduct, which resulted in the conviction of Crosley Green (left) who was later sentenced to death, an absolute travesty of justice not uncommon as I have documented repeatedly.

It all began on April 4, 1989 when Flynn's former girlfriend, Kimberly Hallock called 911 saying she thought her boyfriend had been shot by a a black man with a gun who had hijacked and drove them to a remote Florida citrus grove. Keep in mind, this black guy would've had to steer and shift gears all while he was holding the gun on them. Oh, and after her ex-boyfriend, with his hands tied behind his back, grabbed a gun and shot the assailant, she alone managed to get back into the truck and escape.

Despite a story that kept changing, littered with troubling inconsistencies:
  • Crosley didn't match the original description of the assailant;
  • ex-girlfriend waiting almost an hour to call for help;
  • a truck that was hard to handle because it had a custom gear shift;
  • shoe prints that did not match...that tracked in different direction than testimony indicated;
  • no gun powder residue on Flynn's hands; no shell casings from Flynn's testified to gunshots;
  • no bare footprints or knee prints of either Flynn and Hallock at scene
  • witnesses who later said they were coerced into testifying recanted their testimony
  • a police dog, despite not having an item of Green's to scent upon, somehow connected him to the crime scene
  • no fingerprints or any physical evidence that linked Green to the crime
  • ten alibi witnesses who place Green miles away from the murder,
Crosley Green was arrested and charged with kidnapping, robbery and murder. It took the all-white jury just three hours to convict Crosley Green; the judge sentenced him death.

Crosley Green, top row center, target with a bull's eye...the black spot you focus on.
That's a target with a bull's-eye for Crosley Green. ...His picture is smaller and darker than the other pictures," Harrison said of the photo lineup. "Anybody involved in police investigation and prosecution knows this. ...the position that your eyes are normally drawn to are right in the middle."

"It's a black spot," Green said of the photo. "That's what you focus on, that black spot." [...]
When I went to homicide school ... they told us that this spot is the most likely that someone will pick a picture from," Mark Rixey said of the photo lineup.

"And where exactly is Crosley Green in that--"

"That's Crosley Green right in that spot," Green said, pointing to the second of three images in the top row.

"Anything that strikes you about this lineup?" Moriarty asked Christopher White.

"You can't see the guy in the top middle very well at all," he replied. "Crosley Green's photo is the darkest."

"If you don't specifically know who you're looking for, then that's the spot you will pick nine times out of 10," said Rixey.
And why was Kim Hallock eliminated as a suspect when it's normal procedure to investigate the last people to see victim alive...everyone closest to the scene? No one knows.
That's homicide 101, anybody who is present at the scene of a shooting ... gets their hands tested for gunshot residue," Rixey explained. "That should have been the very first thing that was done. ...That was never done."
Moreover, the Brevard County State's Attorney's Office had a history of pressuring, coercing witnesses into lying. In the 1980s, Brevard County put away three men whose convictions have since been overturned.
They coerced witnesses ... to lie and it's really as though you see -- a deliberate pattern of the state creating evidence to achieve a result that they wanted to achieve and that's what they got," said Jeane Thomas.
In 2009, Green received a reduced sentence (life in prison)  due to an error in sentencing. In total, Crosley Green has spent almost 26 years incarcerated for a crime he did not commit.  Three witnesses recanted their testimony. 
Every witness recanted their story," Moura explained. "And every one of them had reason to be afraid of the police. ...They were squeezed. ...And they were squeezed hard."
As it stands now, undeterred by exculpatory evidence withheld by the prosecution, the recantations of four of the prosecution's star witnesses, not to mention, more inconsistencies than a government's official story, the Florida Attorney General's Office is fighting to uphold Crosley Green's conviction. It says Green failed to meet a filing deadline for his appeal.


Investigators say condemned man not guilty

Former Florida Death Row Inmate Crosley Green Asks Orlando Federal Court To Overturn His Conviction: Crowell & Moring Files Habeas Corpus


Sunday, April 23, 2017

World War III or Perpetual War?

A few years ago, I spoke to a former database administrator for a defense contractor to the CIA from the 1970s to the late 1990s who had to prepare reports for the Joint Chiefs every morning. He said, over the course of that time, there wasn't a day that went by that we weren't at war with someone, somewhere in the world. And this was before 9/11.

Take the philosophical thought experiment, "If a tree falls in a forest and no one is around to hear it, does it make a sound?" and replace it with, "If one nation makes war upon another, and no one reports on it, did that war really happen?" The modern science of mass manipulation through mainstream mesmerizing media and the distortions, lies and fabrications, incessantly pouring out of this media, has ensured that it hasn't, or if it has, it's not even close to what really occurred.  

WWIII is a loaded term. It assumes that these mass murders that humanity calls war are isolated events with distinct beginnings and ends. This simplification obscures the multi-generational chain reactions that lead up to that moment when swords are drawn or missiles fly.
Of course, there is another reason that the public is rarely conscious of these chain reactions. The ruling class has learned long ago that the best way to take a nation to war is to trick them into it."

Remember, proxy wars are still wars.


The Elite, the ‘Great Game’ and World War III
The control of the US, and of global politics, by the wealthiest families of the planet is exercised in a powerful, profound and clandestine manner. This control began in Europe and has a continuity that can be traced back to the time when the bankers discovered it was more profitable to give loans to governments than to needy individuals.

These banking families and their subservient beneficiaries have come to own most major businesses over the two centuries during which they have secretly and increasingly organized themselves as controllers of governments worldwide and as arbiters of war and peace.


Friday, April 14, 2017

Natural Cancer Bullets A - Z

There are many silver bullets that you can use to fight cancer. I will post some of these bullets (alphabetically) along with sources and brief explanations over the next several months.

Acai Berries

Source: Fruits from the Açaí Palm.

Brazilian berry destroys cancer cells in lab, UF study shows
Published today in the Journal of Agricultural and Food Chemistry, the study showed extracts from acai (ah-SAH’-ee) berries triggered a self-destruct response in up to 86 percent of leukemia cells tested, said Stephen Talcott, an assistant professor with UF’s Institute of Food and Agricultural Sciences. “Acai berries are already considered one of the richest fruit sources of antioxidants,” Talcott said. “This study was an important step toward learning what people may gain from using beverages, dietary supplements or other products made with the berries.”


Sources: Aloe Vera Plant, Aloe Vera Juice

Aloe-Emodin Induces Apoptosis in T24 Human Bladder Cancer Cells

AE inhibited cell viability, and induced G2/M arrest and apoptosis in T24 cells. AE increased the levels of Wee1 and cdc25c, and may have led to inhibition of the levels of cyclin-dependent kinase 1 and cyclin B1, which cause G2/M arrest. AE induced p53 expression and was accompanied by the induction of p21 and caspase-3 activation, which was associated with apoptosis. In addition, AE was associated with a marked increase in Fas/APO1 receptor and Bax expression but it inhibited Bcl-2 expression.

Anticancer effect of aloe-emodin on cervical cancer cells involves G2/M arrest and induction of differentiation.
Aloe-emodin inhibited the growth of HeLa cells in a dose-dependent manner at concentrations ranging between 2.5 and 40 micromol/L. The flow cytometric analysis showed that HeLa cells were arrested at the G2/M phase. This effect was associated with the decrease in cyclin A and CDK2, and the increase in cyclin B1 and CDK1. More importantly, the ALP activity was found to be increased by aloe-emodin treatment, and accompanied by the inhibition of PCNA expression. In addition, aloe-emodin suppressed the expression of PKCalpha and c-myc.

Aloe-emodin suppresses prostate cancer by targeting the mTOR complex 2
Our results herein are noteworthy in that prostate cancer cell growth is suppressed by aloe-emodin in vivo. Moreover, the low in vivo toxicity and tumor inhibitory activity of aloe-emodin observed in nude mice suggest that aloe-emodin is an effective chemopreventive agent against prostate cancer (Figure 6A and B). In conclusion, we showed here that mTORC2 is closely associated with prostate cancer cell growth. We also provided clear evidence showing that aloe-emodin effectively suppresses anchorage-independent cell growth and in vivo tumor growth in PC3 cancer cell-bearing nude mice by inhibiting Akt activity. Collectively, these findings support the anticancer efficacy of aloe-emodin through its targeting of mTORC2 for the inhibition of prostate cancer progression.
Protein kinase C involvement in aloe-emodin- and emodin-induced apoptosis in lung carcinoma cell
This study demonstrated aloe-emodin- and emodin-induced apoptosis in lung carcinoma cell lines CH27 (human lung squamous carcinoma cell) and H460 (human lung non-small cell carcinoma cell). Aloe-emodin- and emodin-induced apoptosis was characterized by nuclear morphological changes and DNA fragmentation.

The antiproliferative activity of aloe-emodin is through p53-dependent and p21-dependent apoptotic pathway in human hepatoma cell lines

The aim of this study is to investigate the anticancer effect of aloe-emodin in two human liver cancer cell lines, Hep G2 and Hep 3B. We observed that aloe-emodin inhibited cell proliferation and induced apoptosis in both examined cell lines, but with different the antiproliferative mechanisms.
Aloe-emodin Is a New Type of Anticancer Agent with Selective Activity against Neuroectodermal Tumors
Here we report that aloe-emodin (AE), a hydroxyanthraquinone present in Aloe vera leaves, has a specific in vitro and in vivo antineuroectodermal tumor activity. The growth of human neuroectodermal tumors is inhibited in mice with severe combined immunodeficiency without any appreciable toxic effects on the animals. The compound does not inhibit the proliferation of normal fibroblasts nor that of hemopoietic progenitor cells. The cytotoxicity mechanism consists of the induction of apoptosis, whereas the selectivity against neuroectodermal tumor cells is founded on a specific energy-dependent pathway of drug incorporation. Taking into account its unique cytotoxicity profile and mode of action, AE might represent a conceptually new lead antitumor drug.
Aloe-emodin induced in vitro G2/M arrest of cell cycle in human promyelocytic leukemia HL-60 cells.
Aloe-emodin inhibited cell proliferation and induced G2/M arrest and apoptosis in HL-60 cells. Investigation of the levels of cyclins B1, E and A by immunoblot analysis showed that cyclin E level was unaffected, whereas cyclin B1 and A levels increased with aloe-emodin in HL-60 cells. Investigation of the levels of cyclin-dependent kinases, Cdk1 and 2, showed increased levels of Cdk1 but the levels of Cdk2 were not effected with aloe-emodin in HL-60 cells. The levels of p27 were increased after HL-60 cells were cotreated with various concentrations of aloe-emodin.
Aloe-emodin-induced apoptosis in human gastric carcinoma cells
The purpose of this study was to investigate the anticancer effect of aloe-emodin, an anthraquinone compound present in the leaves of Aloe vera, on two distinct human gastric carcinoma cell lines, AGS and NCI-N87. We demonstrate that aloe-emodin induced cell death in a dose- and time-dependent manner. Noteworthy is that the AGS cells were generally more sensitive than the NCI-N87 cells. Aloe-emodin caused the release of apoptosis-inducing factor and cytochrome c from mitochondria, followed by the activation of caspase-3, leading to nuclear shrinkage and apoptosis.

Aloe-emodin induces in vitro G2/M arrest and alkaline phosphatase activation in human oral cancer KB cells
Aloe-emodin is a natural anthraquinone compound from the root and rhizome of Rheum palmatum. In this study, KB cells were treated with 2.5, 5, 10, 20, and 40 microM aloe-emodin for 1 to 5 days. The results showed that aloe-emodin inhibited cancer cells in a dose-dependent manner. Treatment with aloe-emodin at 10 to 40 microM resulted in cell cycle arrest at G2/M phase. The alkaline phosphatase (ALP) activity in KB cells increased upon treatment with aloe-emodin when compared to controls. This is one of the first studies to focus on the expression of ALP in human oral carcinomas cells treated with aloe-emodin. These results indicate that aloe-emodin has anti-cancer effect on oral cancer, which may lead to its use in chemotherapy and chemo preventment of oral cancer.


Source: Cannabis

Anandamide May Fight Aggressive Skin Cancer
According to the study’s results, anandamide may be involved in a complex mechanism that includes the CB1 receptor, and possibly GPR55 – a cannabinoid receptor in its own right. Although not much is known about GPR55, it is sometimes referred to as the CB3 receptor, because it responds to both endogenous and plant-derived cannabinoids.
The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation
Anandamide was the first brain metabolite shown to act as a ligand of “central” CB1 cannabinoid receptors. Here we report that the endogenous cannabinoid potently and selectively inhibits the proliferation of human breast cancer cells in vitro. Anandamide dose-dependently inhibited the proliferation of MCF-7 and EFM-19 cells with IC50 values between 0.5 and 1.5 μM and 83–92% maximal inhibition at 5–10 μM. The proliferation of several other nonmammary tumoral cell lines was not affected by 10 μM anandamide. The anti-proliferative effect of anandamide was not due to toxicity or to apoptosis of cells but was accompanied by a reduction of cells in the S phase of the cell cycle. ...These data suggest that anandamide blocks human breast cancer cell proliferation through CB1-like receptor-mediated inhibition of endogenous prolactin action at the level of prolactin receptor.

Anti-proliferative and apoptotic effects of anandamide in human prostatic cancer cell lines
ANA induced a decrease of EGFR levels on LNCaP, DU145, and PC3 prostatic cancer cells by acting through cannabinoid CB1 receptor subtype and this leaded to an inhibition of the EGF-stimulated growth of these cells. Moreover, the G1 arrest of metastatic DU145 and PC3 growth was accompanied by a massive cell death by apoptosis and/or necrosis while LNCaP cells were less sensitive to cytotoxic effects of ANA. The apoptotic/necrotic responses induced by ANA on these prostatic cancer cells were also potentiated by the acidic ceramidase inhibitor, N-oleoylethanolamine and partially inhibited by the specific ceramide synthetase inhibitor, fumonisin B1 indicating that these cytotoxic actions of ANA might be induced via the cellular ceramide production. The potent anti-proliferative and cytotoxic effects of ANA on metastatic prostatic cancer cells might provide basis for the design of new therapeutic agents for effective treatment of recurrent and invasive prostatic cancers.
Cannabinoids, Endocannabinoids and Cancer
Cannabinoids exert a number of interesting effects that are dependent on the cell line or tumor type. Synthetic cannabinoids and the endocannabinoid system are implicated in inhibiting cancer cell proliferation and angiogenesis, reducing tumor growth and metastases, and inducing apoptosis.
Anandamide is an endogenous inhibitor for the migration of tumor cells and T lymphocytes
Cell migration is of paramount importance in physiological processes such as immune surveillance, but also in the pathological processes of tumor cell migration and metastasis development. The factors that regulate this tumor cell migration, most prominently neurotransmitters, have thus been the focus of intense investigation. While the majority of neurotransmitters have a stimulatory effect on cell migration, we herein report the inhibitory effect of the endogenous substance anandamide on both tumor cell and lymphocyte migration. ...Using the specific agonist docosatetraenoylethanolamide (DEA), we have observed that the norepinephrine-induced migration of colon carcinoma cells is inhibited by the CB1-R. The SDF-1–induced migration of CD8+ T lymphocytes was, however, inhibited via the CB2-R, as shown by using the specific agonist JWH 133. Therefore, specific inhibition of tumor cell migration via CB1-R engagement might be a selective tool to prevent metastasis formation without depreciatory effects on the immune system of cancer patients.
The endogenous cannabinoid, anandamide, induces cell death in colorectal carcinoma cells
These findings suggest anandamide may be a useful chemopreventive/therapeutic agent for colorectal cancer as it targets cells that are high expressors of COX-2, and may also be used in the eradication of tumour cells that have become resistant to apoptosis.


Sources: Parsley, Celery, Coriander, Licorice, Majoram, Oregano, Rosemary, Tarragon, Citrus, Tea and Wheat.

Breast cancer effectively treated with chemical found in celery, parsley, mouse study suggests
Apigenin, a natural substance found in grocery store produce aisles, shows promise as a non-toxic treatment for an aggressive form of human breast cancer, following a new study. Researchers found apigenin shrank a type of breast cancer tumor that is stimulated by progestin, a synthetic hormone given to women to ease symptoms related to menopause.
Apigenin induces breast cancer cells
Apigenin is a low toxicity and non-mutagenic phytopolyphenol and protein kinase inhibitor. It exhibits anti-proliferating effects on human breast cancer cells. Here we examined several human breast cancer cell lines having different levels of HER2/neu expression and found that apigenin exhibited potent growth-inhibitory activity in HER2/neu-over expressing breast cancer cells but was much less effective for those cells expressing basal levels of HER2/neu
Selective growth-inhibitory, cell-cycle deregulatory and apoptotic response of apigenin in normal versus human prostate carcinoma cells
The growth-inhibitory and apoptotic potential of apigenin was also observed in a variety of prostate carcinoma cells representing different stage and androgen responsiveness. Apigenin may be developed as a promising chemopreventive and/or chemotherapeutic agent against prostate cancer.
Signal pathways involved in apigenin inhibition of growth and induction of apoptosis of human anaplastic thyroid cancer cells
Recently we demonstrated that several flavonoids can inhibit the proliferation of certain human thyroid cancer cell lines. Among the flavonoids tested, apigenin and luteolin are the most effective inhibitors of these tumor cell lines. In the present study, we investigated the signal transduction mechanism associated with the growth inhibitory effect of apigenin, using a human anaplastic thyroid carcinoma cell line, ARO.

5,6-Dichloro-ribifuranosylbenzimidazole- and apigenin-induced sensitization of colon cancer cells to TNF--mediated apoptosis

Here we report that inhibition of CK2 in HCT-116 and HT-29 cells with the use of two specific CK2 inhibitors, 5,6-dichloro-ribifuranosylbenzimidazole (DRB) and apigenin, effected a synergistic reduction in cell survival when used in conjunction with TNF-. Furthermore, there was a demonstrable synergistic reduction in colony formation in soft agar with the use of the same combinations.

Induction of apoptosis by apigenin in leukaemia HL-60 cells
The potency of these flavonoids on these features of apoptosis were in the order of: apigenin>quercetin>myricetin>kaempferol in HL-60 cells treated with 60μM flavonoids. These results suggest that flavonoid-induced apoptosis is stimulated by the release of cytochrome c to the cytosol, by procaspase-9 processing, and through a caspase-3-dependent mechanism. The induction of apoptosis by flavonoids may be attributed to their cancer chemopreventive activity. Furthermore, the potency of flavonoids for inducing apoptosis may be dependent on the numbers of hydroxyl groups in the 2-phenyl group and on the absence of the 3-hydroxyl group. This provides new information on the structure–activity relationship of flavonoids.
Arachidonyl Ethanolamide

Source: Cannabis

Arachidonyl ethanolamide induces apoptosis of uterine cervix cancer cells via aberrantly expressed vanilloid receptor-1
The major finding was that AEA induced apoptosis of CxCa cell lines via aberrantly expressed vanilloid receptor-1, whereas AEA binding to the classical CB1 and CB2 cannabinoid receptors mediated a protective effect. Furthermore, unexpectedly, a strong expression of the three forms of AEA receptors was observed in ex vivo CxCa biopsies.

Sources: wormwood plant

Artemisinin Induces Apoptosis in Human Cancer Cells
Artemisinin is a chemical compound extracted from the wormwood plant, Artemisia annua L. It has been shown to selectively kill cancer cells in vitro and retard the growth of implanted fibrosarcoma tumors in rats. In the present research, we investigated its mechanism of cytotoxicity to cancer cells. ...This rapid induction of apoptosis in cancer cells after treatment with DHA indicates that artemisinin and its analogs may be inexpensive and effective cancer agents.
Effects of artemisinin and its derivatives on growth inhibition and apoptosis of oral cancer cells
Artemisinin is of special biological interest because of its outstanding antimalarial activity. Recently, it was reported that artemisinin has antitumor activity. Its derivatives, artesunate, arteether, and artemeter, also have antitumor activity against melanoma, breast, ovarian, prostate, CNS, and renal cancer cell lines. Recently, monomer, dimer, and trimer derivatives were synthesized from deoxoartemisinin, and the dimers and the trimers were found to have much more potent antitumor activity than the monomers. ...The deoxoartemisinin trimer was found to have greater antitumor effect on tumor cells than other commonly used chemotherapeutic drugs, such as 5-FU, cisplatin, and paclitaxel. Furthermore, the ability of artemisinin and its derivatives to induce apoptosis highlights their potential as chemotherapeutic agents, for many anticancer drugs achieve their antitumor effects by inducing apoptosis in tumor cells.
Transferrin overcomes drug resistance to artemisinin in human small-cell lung carcinoma cells
Multiple drug resistance is a significant problem in small-cell lung cancer (SCLC). Artemisinin (ART) is a natural product used to treat drug-resistant malaria. The drug is effective because the Fe2+ present in infected erythrocytes acts non-enzymatically to convert ART to toxic products. We tested the effects of ART on drug-sensitive (H69) and multi-drug-resistant (H69VP) SCLC cells, pretreated with transferrin (TF) to increase the intracellular Fe2+ level. ...These data indicate the potential use of ART and TF in drug-resistant SCLC.
Dihydroartemisinin induces apoptosis and sensitizes human ovarian cancer cells to carboplatin therapy
The present study was designed to determine the effects of artemisinin (ARS) and its derivatives on human ovarian cancer cells, to evaluate their potential as novel chemotherapeutic agents used alone or in combination with a conventional cancer chemotherapeutic agent, and to investigate their underlying mechanisms of action. Human ovarian cancer cells (A2780 and OVCAR-3), and immortalized non-tumourigenic human ovarian surface epithelial cells (IOSE144), were exposed to four ARS compounds for cytotoxicity testing. The in vitro and in vivo antitumour effects and possible underlying mechanisms of action of dihydroartemisinin (DHA), the most effective compound, were further determined in ovarian cancer cells. ...These effects were also observed in in vivo ovarian A2780 and OVCAR-3 xenograft tumour models. In conclusion, ARS derivatives, particularly DHA, exhibit significant anticancer activity against ovarian cancer cells in vitro and in vivo, with minimal toxicity to non-tumourigenic human OSE cells, indicating that they may be promising therapeutic agents for ovarian cancer, either used alone or in combination with conventional chemotherapy.

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